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Prekallikrein and Kallikrein Inhibitor in Plasma of Patients Affected by Recent Myocardial Infarction

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Vasopeptides

Part of the book series: Advances in Experimental Medicine and Biology ((AEMB,volume 21))

Abstract

Different causes may carry to the shock. The shock credibly has a pathological substrate, that is a damage of the microvessels, particularly capillaries and venules, vasa vasorum included; the sufference of the microvessels concerns mainly the endothelial and basal membrane. Pathogenetically the basic mechanism of the shock is the capillary damage by hypoxia. Hypoxia, in order to establish a capillary damage, must be calibrated in intensity and duration; differently hypoxia, if too important and acute, provokes death following an acute brain damage; if instead too slight, hypoxia does not damage sufficiently the microvessels.

With the support of the National Council of Researches, Rome, Italy.

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© 1972 Plenum Press, New York

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Sicuteri, F., Antonini, F.M., Del Bianco, P.L., Franchi, G., Curradi, C. (1972). Prekallikrein and Kallikrein Inhibitor in Plasma of Patients Affected by Recent Myocardial Infarction. In: Back, N., Sicuteri, F. (eds) Vasopeptides. Advances in Experimental Medicine and Biology, vol 21. Springer, Boston, MA. https://doi.org/10.1007/978-1-4684-7439-8_51

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  • DOI: https://doi.org/10.1007/978-1-4684-7439-8_51

  • Publisher Name: Springer, Boston, MA

  • Print ISBN: 978-1-4684-7441-1

  • Online ISBN: 978-1-4684-7439-8

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