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Discussion of Session

  • Oscar Sudilovsky
  • Henry C. Pitot
  • Lance A. Liotta
Part of the Basic Life Sciences book series (BLSC, volume 57)

Abstract

Moderator (Oscar Sudilovsky): After such interesting presentations I think it is time to concentrate on the mechanistic similarities between tumor promotion and progression. Dr. Pitot, if I interpret him correctly, said that the effect of promoting agents can extend to cells in the stage of progression, while Dr. Slaga expressed the opinion that both processes exist as a continuum. So I would like to start the discussion by asking Dr. Pitot how he would contrast his belief with those of Dr. Slaga.

Keywords

Acute Lymphocytic Leukemia Atrophic Gastritis Viral Oncogene Individual Focus Normal Liver Cell 
These keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.

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References

  1. 1.
    O. Sudilovsky and T. K. Hei: Aneuploid nuclear DNA content in some enzyme-cultured rat liver foci during tumor promotion. Fed. Proc. 42:7 (1983).Google Scholar
  2. 2.
    J. H. Wang, L. Hinrichsen, C. Mansilla and O. Sudilovsky: Hepatocarcinogenesis: Aneuploid nuclear DNA content in enzyme-altered foci (EAF) in rats. Lab. Invest. 58:100A (1988).Google Scholar
  3. 3.
    S. L. Robbins and R. S. Cotran, “The Pathologic Basis of Disease”, 2nd Edition, W. B. Saunders Company, Philadelphia, PA, 1979, p. 182.Google Scholar

Copyright information

© Plenum Press, New York 1991

Authors and Affiliations

  • Oscar Sudilovsky
    • 1
  • Henry C. Pitot
    • 2
  • Lance A. Liotta
    • 3
  1. 1.Case Western Reserve UniversityClevelandUSA
  2. 2.McArdle Laboratory for Cancer ResearchMadisonUSA
  3. 3.National Institutes of HealthBethesdaUSA

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