Mouse Hepatitis Virus 3 Pathogenicity and B and T Lymphotropisms
Viral pathogenicity may be regulated by a host defense mechanism during virus-cell interaction at the immune system level. When the virus is lymphotropic, understanding the pathogenic process of the viral disease is complicated by the virus-lymphocyte interaction which may alter the integrity of the cell and induce a subsequent immunodeficiency.
KeywordsBone Marrow Viral Pathogenicity Cell Depletion Cell Subpopulation Thymic Epithelial Cell
Unable to display preview. Download preview PDF.
- 6.Osmond, D.G., and G.V.J. Nossal. 1974. Differencial on of lymphocytes in mouse bone marrow. II. Kinetics of maturation and renewal of antiglobulin binding cells studied by double labeling. Cell . Immunol. 13: 137.Google Scholar
- 9.Borysiewicz, L.K., and J.G.P. Sissons. 1986. Immune response to virus infected cells. Clin. Immunol. Allergy6: 159.Google Scholar
- 13.Lamontagne, L., C. Dupuy, D. Leray, J.P. Chausseau, and J.M. Dupuy. 1985. Coronavirus-induced immunosuppression: Role of mouse hepatitis virus 3-lymphocyte interaction. Prog. Leuk. Biol. 1: 29.Google Scholar
- 15.Lamontagne, L., and J.M. Dupuy. 1984. Persistent in vitro infection with mouse hepatitis virus type 3 in mouse lymphoid cell lines. Infect. & Immun. 44: 716.Google Scholar
- 20.Levy-Leblond, E., D, and J.M. Dupuy. 1979. Genetic study of mouse sensitivity to MHV3 infection: influence of the H-2 complex. J. Immunol. 112: 1359.Google Scholar
- 21.LePrevost, C., J.L. Virelizier, and J.M. Dupuy. 1975. Immunopathology of mouse hepatitis virus type 3. III. Clinical and virologic observations of a persistent viral infection. J. Immunol.115: 640.Google Scholar