Abstract
For more than 20 years, a controversy has raged between the proponents of two conflicting models of alcoholism. One model, typically referred to as the traditional or disease model (Pattison, Sobell, & Sobell, 1977) assumes that alcoholism is a progressive, irreversible disease which may cause the abstaining alcoholic to experience an irresistible craving for alcohol and the indulgent alcoholic to lose control over alcohol consumption. The etiology of alcoholism, according to this model, is physiological and/or genetic in origin. In contrast, the behavioral model assumes that alcohol consumption is a socially acquired, learned pattern of behavior which occurs as a function of antecedent and consequent events. Such events include situational or environmental factors as well as the alcoholic’s cognitive and affective states.
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Lawson, D.M., Craske, M. (1988). Biological Barriers in the Treatment of Alcoholism. In: Linden, W. (eds) Biological Barriers in Behavioral Medicine. The Plenum Series in Behavioral Psychophysiology. Springer, Boston, MA. https://doi.org/10.1007/978-1-4684-5374-4_2
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DOI: https://doi.org/10.1007/978-1-4684-5374-4_2
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