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Soluble GABA/Benzodiazepine/Barbiturate Receptor Interactions in Mammalian Brain

  • F. Anne Stephenson
  • Richard W. Olsen
Part of the Methodological Surveys in Biochemistry and Analysis book series (MSBA, volume 13)

Abstract

GABA (γ-aminobutyric acid) is now established as the major neurotransmitter in mammalian brain, and mediates its effects via the GABA receptor and an associated chloride ion channel (1]. Further accumulated evidence suggests that some GABA receptors possess modulatory sites for certain centrally active compounds, the benzodiazepines and the barbiturates [1].

Keywords

Sodium Deoxycholate Gaba Receptor Mammalian Brain Affinity Column Chromatography Major Neurotransmitter 
These keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.

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References

  1. 1.
    Olsen, R.W. (1981) J. Neurochem. 37, 1–13.CrossRefGoogle Scholar
  2. 2.
    Stephenson, F.A., Watkins, A.E. & Olsen, R.W. (1982) Eur. J. Biochem. 123, 291–298.CrossRefGoogle Scholar
  3. 3.
    Leeb-Lundberg, F., Snowman, A. & Olsen, R.W. (1980) Proc. Nat. Acad. Sci. 77, 7468–7472.CrossRefGoogle Scholar

Copyright information

© Plenum Press, New York 1984

Authors and Affiliations

  • F. Anne Stephenson
    • 1
    • 2
  • Richard W. Olsen
    • 1
    • 2
  1. 1.Department of BiochemistryImperial CollegeLondonUK
  2. 2.Division of Biomedical SciencesUniversity of CaliforniaRiversideUSA

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