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Purine Metabolites, Uracil and cAMP during Exchange Transfusion

  • H. Manzke
  • H. G. Koke
  • K. Kruse
Part of the Advances in Experimental Medicine and Biology book series (AEMB, volume 165)

Abstract

The metabolism of newborn infants during their first week of life is characterized by an increased breakdown of nucleoproteins leading to transient hyperuricemia and hyperuricosuria which is particularly enhanced in infants with hemolytic diseases. Furthermore, there are physiologically considerable changes in the concentration of calcium in the blood of newborns.

Keywords

Uric Acid Newborn Infant Exchange Transfusion Hemolytic Disease Serum Phosphate Concentration 
These keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.

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References

  1. Brown, D.R., Donavan, E.F., Tsang, R.C., Bobik, C.M., Chen, I.-W., and Johnson, J.R., 1978: Urinary phosphate and cyclic AMP excretion following citrate-induced hypocalcemic stimulation of the neonatal parathyroid glands, J.Pediatr. 93: 842PubMedCrossRefGoogle Scholar
  2. Kreuger, A., 1976: Adenine metabolism during and after exchange transfusions in newborn infants with CPD-Adenine blood, Transfusion 16: 249PubMedCrossRefGoogle Scholar
  3. Manzke, H., Spreter v. Kreudenstein, P., Dörner, K., and Kruse, K., 1980, Quantitative measurements of the urinary •excretion of creatinine, uric acid, hypoxanthine and xanthine, uracil, cyclic AMP and cyclic GMP in healthy newborn infants, Eur. J.Pediatr. 133: 157PubMedCrossRefGoogle Scholar

Copyright information

© Plenum Press, New York 1984

Authors and Affiliations

  • H. Manzke
    • 1
  • H. G. Koke
    • 1
  • K. Kruse
    • 1
  1. 1.University Hospital for ChildrenKielGermany

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