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Abstract

The defining characteristic of bacterial transposons is their ability to move to new loci in the absence of extensive DNA sequence homology. Some, designated IS elements, contain only the genes and sites necessary for their own transposition; other transposons are composites of genes necessary for transposition and genes for auxiliary traits (such as antibiotic resistance or virulence)1. Although the mechanism of transposition is unknown, plausible models involving breakage and reunion2 or replication3,4 have been proposed.

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Berg, D.E. et al. (1981). Transposition of the Inverted Repeats of Tn5 . In: Levy, S.B., Clowes, R.C., Koenig, E.L. (eds) Molecular Biology, Pathogenicity, and Ecology of Bacterial Plasmids. Springer, Boston, MA. https://doi.org/10.1007/978-1-4684-3983-0_36

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  • DOI: https://doi.org/10.1007/978-1-4684-3983-0_36

  • Publisher Name: Springer, Boston, MA

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