Advertisement

Gout Disease. Its Natural History Based on 1,000 Observations

  • A. Rapado
  • J. M. Castrillo
Part of the Advances in Experimental Medicine and Biology book series (AEMB, volume 76B)

Abstract

Gout disease is the most frequent articular metabolic process. When diagnosed it can be corrected by controlling either the hyperuricemia (1) or the articular inflammation (2). This permits an exact control, the earlier clinical complications and poor prognosis having dramatically improved in the last few years.

Keywords

Uric Acid Arterial Hypertension Serum Uric Acid Renal Lithiasis Exact Control 
These keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.

Preview

Unable to display preview. Download preview PDF.

Unable to display preview. Download preview PDF.

References

  1. 1.
    Kelley, W.N.: Pharmacologic approach to the maintenance of urate homeostasis. Nephron, 14:99 1975PubMedCrossRefGoogle Scholar
  2. 2.
    Wallace, S.L.: Colchicine and new antiinflammatory drugs for the treatment of gout. Art.&Rheum. 18:847, 1975CrossRefGoogle Scholar
  3. 3.
    Kuzel1, W.C., Schafferzîck, R.W., Naugler, W.E., Koets, P., Mankle, E.A., Brown, B. and Champlin, B.: Some observations on 520 patients of gout. J.chron.Dis. 2:645, 1955CrossRefGoogle Scholar
  4. 4.
    Seze, S. and Ryckwaert, A.: La goutte. L’ Expansion, ed. Paris 1960, p. 67.Google Scholar
  5. 5.
    Grahame, R. and Scott, J.T.: Clinical survey of 354 patients with gout. Ann.rheum.Dis. 29:461, 1970PubMedCrossRefGoogle Scholar
  6. 6.
    Kelgre, J.H.: The epidemiology of chronic diseases. Blackwell, Oxford, 1963, p. 104Google Scholar
  7. 7.
    Rapado, A.: Allopurinol in thiazide-induced hyperuricemia. Ann.rheum.Dis. 25:660, 1966PubMedGoogle Scholar
  8. 8.
    Hadler, N.M., Frank, W.A., Bress, N.M. and Robinson, D.R.: Acute polyarticular gout. Am.J.Med. 56:715, 1974PubMedCrossRefGoogle Scholar
  9. 9.
    Gutman, A.B.: The past four decades of progress in the knowledge of gout, with an assessment of the present status. Art. & Rheum. 16:431, 1973CrossRefGoogle Scholar
  10. 10.
    Rapado, A., Herrera, J.L., Jiménez, M. and Rodriguez, J.L.: Cushing iatrogénico y gota corticodependiente. Rev.clin.esp. 98:275, 1965Google Scholar
  11. 11.
    Mikkelsen, W.M., Dodge, H.J. and Valkenburg, H.: The distribution of serum uric acid values in a population unselected as to gout and hyperuricemia. Am.J.Med. 39:242, 1965PubMedCrossRefGoogle Scholar
  12. 12.
    Healy, L.A.: Epidemiology of hyperuricemia. Art.& Rheum. 18: 709, 1975CrossRefGoogle Scholar
  13. 13.
    Rapado, A.: Relationship between gout and arterial hypertension. In: Purine metabolism in man. vol. 41 B, ed. by O. Sperling, A. de Vries and J.B. Wyngaarden. Plenum Pub. Corp. New York 1974, p. 451Google Scholar
  14. 14.
    Cifuentes, L., Rapado, A., Abehsera, A., Traba, M.L. and Cortés, M.: Uric acid lithiasis and gout. In: Urinary Calculi, ed. by L. Cifuentes, A. Rapado and A. Hodgkinson. Karger, Basel 1973, p. 115.Google Scholar
  15. 15.
    Frank, M., Lazebnik, J. and de Vries, A.: Uric acid lithiasis. A study of six hundred and twenty-two patients. Urol.int. 25: 32, 1970PubMedCrossRefGoogle Scholar
  16. 16.
    Smyth, C.J.: Disorders associated with hyperuricemia. Art.& Rheum. 18:713, 1975CrossRefGoogle Scholar
  17. 17.
    Rapado, A.: Gout and saturnism. New Eng.J.Med. 281:851, 1969PubMedGoogle Scholar
  18. 18.
    Seegmiller, J.E.: Genetic considerations of gout. Art.&Rheurru 18:743, 1975CrossRefGoogle Scholar
  19. 19.
    Lanese, R.R., Gresham, G.E. and Keller, M.D.: Behavioural and physiological characteristics in hyperuricemia. J.A.M.A. 207: 1878, 1969PubMedCrossRefGoogle Scholar
  20. 20.
    O’Duffy, J.D., Hunder, G.G. and Kelly, P.K.: Decreasing prevalence of tophaceous gout. Mayo Clinic Proc. 50:227, 1975Google Scholar
  21. 21.
    Phelps, P., Steele, A.D. and McCarthy, D.J.: Compensated polarized light microscopy: Identification of crystals in synovial fluids from gout and pseudogout. J.A.M.A. 203:166, 1968CrossRefGoogle Scholar
  22. 22.
    Wyngaarden, J.B.: Metabolic defects of primary hyperuricemia and gout. Am.J.Med. 56:651, 1974PubMedCrossRefGoogle Scholar
  23. 23.
    Barlow, K.A. and Beilin, L.J.: Renal disease in primary gout. Quar.J.Med. 37:79, 1968Google Scholar
  24. 24.
    Berger, L. and Yu, T.F.: Renal function in gout. IV. An analysis of 524 gouty patients including long-term follow-up studies. Am.J.Med. 59:605, 1975PubMedCrossRefGoogle Scholar
  25. 25.
    Wiedeman, E., Rose, H.G. and Schwartz, E.: Plasma lipoproteins, glucose tolerance and insulin response in primary gout. Am.J. Med. 53:299, 1972CrossRefGoogle Scholar
  26. 26.
    Yu, T.F., Beilis, E. and Yip, L.C.: Overproduction of uric acid in primary gout. Art.& Rheum. 18:695, 1975CrossRefGoogle Scholar

Copyright information

© Plenum Press, New York 1977

Authors and Affiliations

  • A. Rapado
    • 1
  • J. M. Castrillo
    • 1
  1. 1.Unidad MetabólicaFundación Jiménez Díaz and Universidad AutónomaMadrid-3Spain

Personalised recommendations