Abstract
There is no doubt that the tumor growing in the host finds itself under conditions of severe hypoxia (Vaupel, 1974) due to scarce O2 supply. It is likely that hypoxia would prevent the respiration of cancer cells from fulfilling in vivo its normal physiological functions.
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References
Shapot V.S. Adv.Cancer Res. 15, 253–286, 1972.
Shapot V.S., Blinov V.A. Cancer Res. 34, 1827–1832, 1974.
Vaupel P. Atemgaswechsel und Glucosestoffwechsel von Implantationstumoren (DS-Carcinosarcom) in vivo, 1974.
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© 1976 Plenum Press, New York
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Shapot, V.S. (1976). Studies on the Ability of the Tumor Respiration in Vivo to Fulfill its Physiological Functions. In: Grote, J., Reneau, D., Thews, G. (eds) Oxygen Transport to Tissue — II. Advances in Experimental Medicine and Biology, vol 75. Springer, Boston, MA. https://doi.org/10.1007/978-1-4684-3273-2_68
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DOI: https://doi.org/10.1007/978-1-4684-3273-2_68
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