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Adipose Tissue and Hemorrhagic Shock

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Part of the book series: Advances in Experimental Medicine and Biology ((AEMB,volume 33))

Abstract

The average man has around 15 kg of adipose tissue and as much as 90% of this may be triglycerides. As a consequence, adipose tissue may yield close to the theoretical 9.4 cal/g of pure lipid. The adipose tissue is then by far the largest energy source of the body. It is therefore obvious that it must be of great interest to find out how the adipose tissue reacts during different forms of trauma. There have been a limited number of studies on lipid metabolism, including changes in the plasma FFA level, during trauma. These studies have indicated that there is an increased mobilization of FFA from adipose tissue (Wadström, 1959, Stoner, 1962). Therefore the question has been raised whether high plasma FFA levels produced by elevated mobilization from adipose tissue may be harmful to the individual. It has been argued that heart failure may partly be due to oxidation of fat, since this requires more oxygen than carbohydrate oxidation. Furthermore, high plasma levels of FFA may be the cause of lung embolism.

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© 1973 Plenum Press, New York

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Rosell, S., Sándor, P., Kovách, A.G.B. (1973). Adipose Tissue and Hemorrhagic Shock. In: Kovách, A.G.B., Stoner, H.B., Spitzer, J.J. (eds) Neurohumoral and Metabolic Aspects of Injury. Advances in Experimental Medicine and Biology, vol 33. Springer, Boston, MA. https://doi.org/10.1007/978-1-4684-3228-2_32

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  • DOI: https://doi.org/10.1007/978-1-4684-3228-2_32

  • Publisher Name: Springer, Boston, MA

  • Print ISBN: 978-1-4684-3230-5

  • Online ISBN: 978-1-4684-3228-2

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