Abstract
In 1961, at the Symposium on shock in Stockholm, we presented evidence of persistent renal vasoconstriction in and following hemorrhagic shock which was suggested to take place on the preglomerular level (1). However, total renal blood flow was not so much reduced that hypoxia resulted. This was concluded from measurements of oxygen saturations of cortical blood in the low pressure phase of shock (2).
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References
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© 1970 Plenum Press, New York
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Kramer, K. (1970). Renal Failure in Hemorrhagic Shock. In: Bertelli, A., Back, N. (eds) Shock: Biochemical, Pharmacological, and Clinical Aspects. Advances in Experimental Medicine and Biology, vol 9. Springer, Boston, MA. https://doi.org/10.1007/978-1-4684-3201-5_2
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DOI: https://doi.org/10.1007/978-1-4684-3201-5_2
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