Abstract
For reasons explained in the Introduction, the model must accommodate new variant populations produced through random mutation (which will appear initially in a single infected cell), possible lineage-extinction, and heterogeneous replication. Multiple genomes, PIT types, and some kind of stochastic process are inevitable components.
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Wick, W.D., Yang, O.O. (2013). The HIV Infection Model. In: War in the Body. Springer, New York, NY. https://doi.org/10.1007/978-1-4614-7294-0_2
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