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Alterations in Fatty Acid Composition of Phospholipids in Heart Muscle Induced by Various Forms of Stress

  • S. Gudbjarnason
  • A. Gudmundsdóttir
  • V. E. Benediktsdóttir
Part of the Developments in Cardiovascular Medicine book series (DICM, volume 46)

Abstract

Stress is considered one of the pathogenic factors in development of coronary artery disease. Increased sympathetic activity and elevated plasma levels of catecholamines influence markedly cardiac function and myocardial metabolism. Changes in function of the heart induced by exogenous or endogenous catecholamines during stress or significant changes in living conditions could modify composition of membrane lipids during the process of adaptation. Catecholamines have been shown to affect various aspects of glycerolipid metabolism (1).

Keywords

Linoleic Acid Fatty Acid Composition Arachidonic Acid Ventricular Fibrillation Docosahexaenoic Acid 
These keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.

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References

  1. 1.
    Emilsson, A. and Gudbjarnason, S. Reversible alterations in fatty acid profile of glycerophospholipids in rat heart muscle induced by repeated norepinephrine administration. Biochim. Biophys. Acta 730; 1–6, 1983CrossRefGoogle Scholar
  2. 2.
    Gudmundsdóttir, A. and Gudbjarnason, S. Neonatal changes in fatty acid profile of phospholipids in rat heart muscle. Biochim. Biophys. Acta 752; 284–290, 1983.PubMedGoogle Scholar
  3. 3.
    Gudbjarnason, S., Emilsson, A. and Gudmundsdóttir, A. Fatty acid composition of phospholipids of heart muscle in relation to age, dietary fat and stress. In: H. Peeters, G.A. Gresham and R. Paoletti (ed). Arterial Pollution.Plenum Publ. Corp. 1983, pp. 115–124.Google Scholar
  4. 4.
    Hirata, F. and Axelrod, I. Phospholipid methylation and biological transmission. Science 209: 1082–1090, 1980.PubMedCrossRefGoogle Scholar
  5. 5.
    Daves, G.S. Foetal and Neonatal Physiology. Year Book, Medical Publishers, Inc., Chicago, 1969.Google Scholar
  6. 6.
    Gudbjarnason, S., Whitehurst, V., Simental, F. and Balazs, T. Alterations in fatty acid composition of phospholipids in rat heart muscle during weight loss and increased resistance to cardiotoxicity of isoproterenol. The Toxicologist 4: pp. 77, 1984.Google Scholar
  7. 7.
    Johnson, G.L., El-Hage, A., Ehrreich, S.I., Gudbjarnason, S., Whitehurst, V. and Balazs, T. Effects of benoxaprofen, diethylcarbamazine or nordihydroguaiaretic acid on isoproterenol induced ventricular fibrillation in rats. Federation Proceedings 43: pp. 742, 1984.Google Scholar
  8. 8.
    Johnson, G.L., El-Hage, A., Ehrreigh, S.I., Gudbjarnason, S., Whitehurst, V. and Balazs, T. A possible role of leukotrienes in isoproterenol induced ventricular fibrillation. In “Prostaglandins and Leukotrienes ’84” Washington D.C., Abstract 251, 1984.Google Scholar

Copyright information

© Martinus Nijhoff Publishing, Boston 1985

Authors and Affiliations

  • S. Gudbjarnason
    • 1
  • A. Gudmundsdóttir
    • 1
  • V. E. Benediktsdóttir
    • 1
  1. 1.Science InstituteUniversity of IcelandReykjavikIceland

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