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The Adrenal Gland pp 313–339Cite as

Bartter’s Syndrome

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Part of the book series: Clinical Surveys in Endocrinology ((CSED,volume 2))

Abstract

In the early 1960s Bartter et al.1,2 described a new syndrome characterized by hypokalemic alkalosis, hyperaldosteronism, hyperreninemia, hyperplasia of the juxtaglomerular apparatus, normal blood pressure, and decreased pressor responsiveness to intravenous infusion of angiotensin II. Despite its rarity, Bartter’s syndrome has generated considerable interest owing to the fact that much has been learned by studying this disorder, regarding the role of the renal tubules in the transport of several ions. While considerable controversy exists regarding the nature of the fundamental defect in Bartter’s syndrome, several recent discoveries have narrowed the search for the missing piece in the elegant puzzle that is Bartter’s syndrome. The heterogeneity in the clinical as well as the biochemical expression of this syndrome has led to a heightened awareness of the disorder.3

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Authors and Affiliations

  1. Division of Endocrinology and Metabolism, Department of Medicine, Cook County Hospital, Chicago, Illinois, USA

    C. R. Kannan M.D. (Chairman, Associate Professor of Medicine)

  2. Rush-Presbyterian - St. Luke’s Medical Center, Chicago, Illinois, USA

    C. R. Kannan M.D. (Chairman, Associate Professor of Medicine)

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© 1988 Plenum Publishing Corporation

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Kannan, C.R. (1988). Bartter’s Syndrome. In: The Adrenal Gland. Clinical Surveys in Endocrinology, vol 2. Springer, Boston, MA. https://doi.org/10.1007/978-1-4613-1001-3_7

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  • DOI: https://doi.org/10.1007/978-1-4613-1001-3_7

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