Abstract
Intracellular cholesterol accumulation in the arterial wall and the proliferation of intimal smooth muscle cells are the hallmarks of atherogenesis. Humoral fluid-phase interactions between arterial endothelial and smooth muscle cells have been studied in vivo and in vitro in an attempt to delineate whether these cells communicate in the regulation of intimal hyperplasia and cholesterol and lipoprotein metabolism in arterial smooth muscle cells.
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© 1988 Plenum Press, New York
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Hajjar, D.P., Marcus, A.J., Pomerantz, K.B., Hajjar, K.A. (1988). Arterial Cell Interactions: Mechanistic Studies Related to Eicosanoid and Growth Factor-Induced Alterations in Cholesterol Metabolism. In: Malmendier, C.L., Alaupovic, P. (eds) Eicosanoids, Apolipoproteins, Lipoprotein Particles, and Atherosclerosis. Advances in Experimental Medicine and Biology, vol 243. Springer, Boston, MA. https://doi.org/10.1007/978-1-4613-0733-4_5
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DOI: https://doi.org/10.1007/978-1-4613-0733-4_5
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