Abstract
Copper, in trace quantities, is required by all living organisms to maintain proper cellular function. In excess, copper is extremely toxic due to the chemical reactivity of the element. To maintain the necessary balance between required and toxic quantities of copper, living organisms have evolved with mechanisms to conserve copper when the dietary level is low and rid the body of excess when necessary. The following discussion describes our current knowledge of the mechanisms involved in the regulation of copper homeostasis.
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Evans, G.W., Johnson, W.T. (1982). Copper Homeostasis. In: Sorenson, J.R.J. (eds) Inflammatory Diseases and Copper. Experimental Biology and Medicine, vol 2. Humana Press. https://doi.org/10.1007/978-1-4612-5829-2_2
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DOI: https://doi.org/10.1007/978-1-4612-5829-2_2
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