The term “spinal cord compression” is widely used in clinical practice to indicate the pathological and clinical entity caused by an expanding lesion within the spinal canal. There are a great number of causes of compression (Table 10.1). The pressure effect could be on the spinal cord and/or the nerve roots emerging from the cord. A rapidly expanding lesion will cause an “acute” clinical presentation whereas the symptoms of a slow-growing benign tumour will evolve over a much longer period. Occasionally, “chronic” compression can lead to an acute course usually due to sudden rapid expansion or ischaemia from vascular occlusion. The neurological features are determined by the rate of expansion of the lesion and the spinal level involved. Careful neurological assessment will localize the anatomical site of compression. There are well recognized patterns or “syndromes” of clinical presentation. Certain diseases display symptoms and signs due to involvement of other systems. Early diagnosis results in a better prognosis and functional outcome. This can be achieved by combining good clinical knowledge with careful assessment of patients followed by appropriate investigations.
KeywordsNerve Root Spinal Canal Cord Compression Sensory Level Radicular Pain
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- Brown-Sequard CE (1850) De la transmission crosse des impressions sensitives par la moelle epiniere. CR Biol 2:33Google Scholar
- Friehs GM, Schrottner 0, Pendl G (1995) Evidence for segregated pain and temperature conduction within the spinothalamic tract. J Neurosurg 83:8–12Google Scholar
- Hayashi N, Green BA, Gonzales-Carvajal M et al. (1983) Local blood flow, oxygen tension and oxygen consumption in the rat spinal cord. Part 2. J Neurosurg 58:526–530Google Scholar
- Lhermitte J, Bollack, Nicholas M (1924) Les douleurs a type de decharge electrique consecutives a la flexion cephalique dans la sclerose en placques; sclerose multiple Rev Neurol 31:56–62Google Scholar
- Raynor RB (1969) Papilledema associated with tumours of the spinal cord. Neurology Minneapolis 19:700Google Scholar