Cytoplasmically inherited male sterility (CMS) the maternally inherited failure to produce or shed functional pollen, has been reported in a wide range of plant species. In many cases, the genetic basis for this trait has been traced to the mitochondrial genome. Although male fertility is affected in all cases, the developmental features are varied, as are the molecular mechanisms that produce them. While mitochondrial genomes encode CMS, nuclear restorer-of-fertility genes can suppress or compensate for the expression of mitochondrial CMS genes, thereby conditioning male fertility. Molecular mechanisms of fertility restoration are also varied. CMS and fertility restoration provide a unique set of molecular - genetic tools for the investigation of interactions between nuclear and mitochondrial genetic systems.
Access this chapter
Tax calculation will be finalised at checkout
Purchases are for personal use only
Preview
Unable to display preview. Download preview PDF.
Author information
Authors and Affiliations
Editor information
Editors and Affiliations
Rights and permissions
Copyright information
© 2004 Springer
About this chapter
Cite this chapter
Chase, C.D., Gabay-Laughnan, S. (2004). Cytoplasmic Male Sterility and Fertility Restoration by Nuclear Genes. In: Daniell, H., Chase, C. (eds) Molecular Biology and Biotechnology of Plant Organelles. Springer, Dordrecht. https://doi.org/10.1007/978-1-4020-3166-3_22
Download citation
DOI: https://doi.org/10.1007/978-1-4020-3166-3_22
Publisher Name: Springer, Dordrecht
Print ISBN: 978-1-4020-2713-0
Online ISBN: 978-1-4020-3166-3
eBook Packages: Springer Book Archive