It is usually much easier to design chemotherapy for a disease when the origins and aetiology of the disease is known, but this is not possible in the case of cancer. Some affirm that because there are many types of cancer, presenting many different aetiologies, then there must be as many causes as there are types. Others, using the principle of Occam’s razor, consider that there must be an overall single cause of cancer and that the different aetiologies are due to the nature of the tissue in which the disease originates. It is now generally accepted that there must be a single or a very small number of different causes of cancer.
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13.7 References and Further Reading
- Baserga, R. (1971). The Cell Cycle and Cancer, Marcel Dekker, New YorkGoogle Scholar
- Boesen, E. and Davis, W. (1969). Cytotoxic Drugs in the Treatment of Cancer. Edward Arnold, LondonGoogle Scholar
- Brule, G., Eckhardt, S. J., Hall, T. C. and Winkler, A. (1973). Drug Therapy of Cancer. World Health Organisation, GenevaGoogle Scholar
- Cline, M. J. and Haskell, C. M. (1975). Cancer Chemotherapy. Saunders, PhiladelphiaGoogle Scholar
- Edwards, D. I. (1979). Cancer chemotherapy-new approaches. In Companion to the Life Sciences, Vol. 2 (S. Day, ed.). Van Nostrand-Rheinhold, New York (in press)Google Scholar
- Graham, F. L. and Whitamore, G. F. (1970). Studies in mouse L-cells on the incorporation of 1-β-D-arabinofuranosylcytosine into DNA and on inhibition of DNA polymerase by 1-β-D-arabinofuranosyl cytosine-5-triphosphate. Cancer Res., 30, 2636–45Google Scholar
- Nakayama, H. and Hanawalt, P. (1975). Sedimentation analysis of deoxyribonucleic acid from thymine-starved Escherichia coli. J. Bact., 121, 537–47Google Scholar
- Passwater, R. A. (1973). Cancer-new drections. Int. Lab., 10–19, July/AugustGoogle Scholar
- Pratt, W. B. (1973). Fundamentals of Chemotherapy. Oxford University Press, LondonGoogle Scholar