Role of the Blood—Brain Barrier in Immunopathogenesis of Experimentally Induced Autoimmune Demyelination
Experimental allergic encephalomyelitis (EAE) has been studied as experimental model for human primary demyelinating diseases such as multiple sclerosis. There are a number of experimental data indicating altered function of the blood-brain barrier (BBB) during the acute and chronic-relapsing forms of EAE (Bradbury, 1989).
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