Aetiology, Classification and Clinical Characteristics of Esotropia in Infancy

  • Brian Harcourt
Part of the Wenner-Gren Center International Symposium Series book series (WGS)

Abstract

Normal oculomotor function depends basically upon mechanisms which control ocular versions and vergences. Failure of normal versions is manifest clinically in gaze palsies; failure of appropriate vergences may give rise to non-paralytic strabismus. Failures of either or both may cause nystagmus. Even if the supranuclear systems and the ocular muscles develop normally, incomitant strabismus will ensue if there are abnormalities in ocular motor nerve functions or mechanical defects limiting ocular rotations. In considering the relationship between the development of oculomotor control and the incidence of strabismus, particular attention must therefore be concentrated on the vergence mechanisms and on the developmental factors which can cause paralytic strabismus of neurogenic or mechanical origin.

Keywords

Retina Adduct Iodide Dura Thalidomide 

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Copyright information

© The Wenner-Gren Center 1988

Authors and Affiliations

  • Brian Harcourt

There are no affiliations available

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