Conclusions
Our data demonstrate a new ligand specificity of TLR2, since LcrV is the first known secreted and non-lipidatedvirulence-associated protein of a Gram-negative bacterium utilizing TLR2 for cell activation. We conclude that yersiniae might exploit host innatepattern recognition molecules and defense mechanisms to evade the host immune response.
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© 2004 Kluwer Academic Publishers
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Sing, A. et al. (2004). Mechanisms of Yersinia enterocolitica Evasion of the Host Innate Immune Response by V Antigen. In: Skurnik, M., Bengoechea, J.A., Granfors, K. (eds) The Genus Yersinia. Advances in Experimental Medicine and Biology, vol 529. Springer, Boston, MA. https://doi.org/10.1007/0-306-48416-1_33
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DOI: https://doi.org/10.1007/0-306-48416-1_33
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