Phosphate and Mineral Metabolism pp 241-252 | Cite as
Changes Following Parathyroidectomy in Hemodialyzed Patients
Abstract
Secondary hyperparathyroidism (HPT) remains a frequent complication of chronic renal failure (CRF) despite a better understanding of the factors responsible for its development (1). A prolonged survival of the patients by dialysis results in a growing number of uremics who present with severe HPT responsible not only for major bone lesions but also for an impairment of different vital functions (2). The metabolites of vitamin D such a 1,25 dihydroxycholecalciferol that can play a preventive role in renal osteodystrophy at an early stage of chronic renal failure (3) are inconsistently effective and difficult to handle during severe HPT (4). Thus, treatment by parathyroidectony (PTX) appears sometimes to be required (5).
Keywords
Chronic Renal Failure Calcification Rate Renal Osteodystrophy Osteitis Fibrosa Subtotal ParathyroidectomyPreview
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