Modulation of Extracellular Kynurenic Acid Content by Excitatory Amino Acids in Primary Cultures of Rat Astrocytes

  • L. Curatolo
  • C. Caccia
  • C. Speciale
  • L. Raimondi
  • M. Cini
  • M. Marconi
  • A. Molinari
  • R. Schwarcz
Part of the Advances in Experimental Medicine and Biology book series (AEMB, volume 398)

Abstract

Kynurenines, i.e. tryptophan metabolites along the kynurenine pathway, have recently received attention because of their possible role in the immunocompromised brain and in the pathogenesis of human neurodegenerative diseases (Stone, 1993). In particular, high levels of the endogenous excitotoxin quinolinic acid and low levels of the excitatory amino acid (EAAs) antagonist kynurenate (KYNA) have been speculatively linked to the occurrence of seizures and nerve cell death.

Keywords

Excitatory Amino Acid Kynurenic Acid Kynurenine Pathway Concentration Of500 Primary Astrocyte Culture 
These keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.

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References

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Copyright information

© Plenum Press, New York 1996

Authors and Affiliations

  • L. Curatolo
  • C. Caccia
    • 1
  • C. Speciale
    • 1
  • L. Raimondi
    • 1
  • M. Cini
    • 1
  • M. Marconi
    • 1
  • A. Molinari
    • 1
  • R. Schwarcz
    • 2
  1. 1.Pharmacia, R&D-CNSNerviano, MilanoItaly
  2. 2.Maryland Psychiatric Research CenterUniversity of Maryland School of MedicineBaltimoreUSA

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