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A Case of False Foster-Kennedy Syndrome

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Part of the book series: Advances in Visual Science and Eye Diseases ((AVSED,volume 2))

Abstract

The etiology of Foster-Kennedy syndrome, also known as Kennedy’s syndrome or basilar frontal lobe syndrome, involves space-occupying lesions at the basilar part of the frontal lobe, such as abscess, hemangioma, meningioma of the sphenoid ridge, carotid atherosclerosis, arachnoiditis, and brain trauma. In typical Foster-Kennedy syndrome cases, unilateral tumor in the anterior cranial fossa compresses the ipsilateral optic nerve, resulting in ipsilateral optic atrophy, and meanwhile space-occupying lesions induce intracranial hypertension, leading to contralateral optic disc edema. The patient in the case below suffered from Foster-Kennedy syndrome with optic disc edema in one eye and a pale optic disc in the other. What is the mechanism behind this condition?

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References

  1. Acebes X, Arruga J, Acebes JJ, et al. Intracranial meningiomatosis causing Foster Kennedy syndrome by unilateral optic nerve compression and blockage of the superior sagittal sinus. J Neuroophthalmol. 2009;29(2):140–2.

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  2. Bansal S, Dabbs T, Long V. Pseudo-Foster Kennedy syndrome due to unilateral optic nerve hypoplasia: a case report. J Med Case Rep. 2008;2(1):1–2.

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© 2019 Springer Nature Singapore Pte Ltd. & People's Medical Publishing House, PR of China

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Xie, X., Fan, N., Wang, N. (2019). A Case of False Foster-Kennedy Syndrome. In: Wang, N., Liu, X., Fan, N. (eds) Optic Disorders and Visual Field. Advances in Visual Science and Eye Diseases, vol 2. Springer, Singapore. https://doi.org/10.1007/978-981-13-2502-1_57

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  • DOI: https://doi.org/10.1007/978-981-13-2502-1_57

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  • Publisher Name: Springer, Singapore

  • Print ISBN: 978-981-13-2501-4

  • Online ISBN: 978-981-13-2502-1

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