Calcium Transport and Intracellular Calcium Homeostasis

  • Danielle Pansu
  • Felix Bronner
Conference proceedings

Part of the NATO ASI Series book series (volume 48)

Table of contents

  1. Front Matter
    Pages I-XXII
  2. Cellular Calcium Entry — Calcium Channels

    1. Front Matter
      Pages 1-1
    2. J. C. J. Saunders, L. C. Isaacson
      Pages 27-34
    3. F. Scheffauer, M. Canonaco-Friedrich, H.-G. Knaus, J. Striessnig, C. Zech, G. Zernig et al.
      Pages 35-48
    4. Gregory J. Kaczorowski, John P. Felix, Maria L. Garcia, V. Frank King, Judith L. Shevell, Robert S. Slaughter
      Pages 59-66
  3. Calcium Extrusion — The Ca ATPases

  4. Calcium Extrusion — The Na/Ca Exchanger

    1. Front Matter
      Pages 103-103
    2. John P. Reeves, Diane C. Ahrens, Joo Cheon, John T. Durkin
      Pages 105-121
    3. M. P. Blaustein, S. Bova, X.-J. Yuan, W. F. Goldman
      Pages 123-132
  5. Regulation and Regulatory Role of Intracellular Calcium

  6. Epithelial Calcium Transport

  7. Calcium-Binding Proteins Characteristics and Structure

  8. The Calbindins — Gene Structure and Function

    1. Front Matter
      Pages 331-331
    2. E. Muir, M. Harding, P. Wilson, D. E. M. Lawson
      Pages 333-338
    3. S. Christakos, A. M. Iacopino, H. Li, S. Lee, R. Gill
      Pages 339-346
    4. M. Thomasset, N. Lomri, F. L’Horset, A. Bréhier, J.-M. Dupret, C. Perret
      Pages 347-354
  9. Defects of Calcium Transport

    1. Front Matter
      Pages 375-375
    2. Uta Hennessen, Bernard Lacour, Tilman B. Drüeke
      Pages 393-400
    3. H. S. Cross, E. Lehner, N. Fratzl-Zelman, K. Klaushofer, M. Peterlik
      Pages 401-408
    4. T. J. B. Simons
      Pages 409-415

About these proceedings


The crucial role played by calcium as a cellular messenger has become increasingly evident, as has the recognition that cells spend much energy in maintaining the cytosolic concentration of this cation both constant and low. It is thought they do this to avoid precipitating phosphate, needed as a source of bond energy and to modulate protein structure. Moreover, since calcium that does enter the cell must be disposed with, processes that utilize calcium have evolved, e.g. secretion, contraction, signaling, to name just some. New knowledge concerning the processes of cellular calcium entry, extrusion and the fate of intracellular calcium has accumulated in recent years. Much has also been learned about calcium transport by and across epithelial cells. It seems logical to think that the processes of calcium entry, extrusion and intracellular handling are similar in all cells. We have therefore assembled in one volume overviews and research reports of transport and cellular calcium regulation so as to explore similarities and differences between cells that utilize calcium for metabolic purposes and those whose primary function is transport.


ATP Calbindin Calcium Calciumtransport Diabetes G proteins Kalzium Vitamin D homeostasis proteins

Editors and affiliations

  • Danielle Pansu
    • 1
  • Felix Bronner
    • 2
  1. 1.Ecole Pratique des Hautes Etudes and INSERM U 45Hôpital E. HerriotLyon Cedex 03France
  2. 2.Dept. of Biostructure and FunctionUniversity of Connecticut, Health CenterFarmingtonUSA

Bibliographic information

  • DOI
  • Copyright Information Springer-Verlag Berlin Heidelberg 1990
  • Publisher Name Springer, Berlin, Heidelberg
  • eBook Packages Springer Book Archive
  • Print ISBN 978-3-642-83979-5
  • Online ISBN 978-3-642-83977-1
  • Buy this book on publisher's site