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The Diabetic Pancreas

  • Bruno W. Volk
  • Edward R. Arquilla

Table of contents

  1. Front Matter
    Pages i-xxiv
  2. Bruno W. Volk, Klaus F. Wellmann
    Pages 1-16
  3. Claes Hellerström, Ingemar Swenne
    Pages 53-79
  4. Enrico Solcia, Carlo Capella, Luciana Usellini, Roberto Fiocca, Fausto Sessa
    Pages 107-115
  5. Bruno W. Volk, Klaus F. Wellmann
    Pages 117-125
  6. Lennart Boquist, Stefan O. Emdin
    Pages 127-169
  7. Lelio Orci, Alain Perrelet
    Pages 171-212
  8. Michael L. McDaniel, Jerry R. Colca, Nirmala Kotagal, Paul E. Lacy
    Pages 213-231
  9. Bruno W. Volk, Klaus F. Wellmann
    Pages 233-263
  10. Bruno W. Volk, Klaus F. Wellmann
    Pages 265-273
  11. Bruno W. Volk, Klaus F. Wellmann
    Pages 275-297
  12. Bruno W. Volk, Klaus F. Wellmann
    Pages 299-318
  13. Bruno W. Volk, Klaus F. Wellmann
    Pages 319-325
  14. Bruno W. Volk, Klaus F. Wellmann
    Pages 327-336
  15. Willy Gepts, Philip M. LeCompte
    Pages 337-365
  16. Bruno W. Volk, Klaus F. Wellmann
    Pages 367-384
  17. Arthur A. Like
    Pages 385-413
  18. Albert Y. Chang, Arthur R. Diani
    Pages 415-438
  19. John E. Craighead
    Pages 439-466
  20. Günter Klöppel
    Pages 467-492
  21. Edward R. Arquilla, David P. Stenger
    Pages 493-512
  22. Orion D. Hegre
    Pages 513-542
  23. Werner Creutzfeldt
    Pages 543-586
  24. M. Michael Wolfe, James E. McGuigan
    Pages 587-615
  25. Back Matter
    Pages 617-628

About this book

Introduction

Since the publication of the first edition of The Diabetic Pancreas in 1977, much progress has been made in various areas of diabetes research. While only a rela­ tively short while ago diabetes was considered a single disease, in more recent years it has become apparent that it is a heterogeneous group of disorders, all of which are characterized by a decreased tolerance of carbohydrates and most of which have a genetic basis, although the genetic types vary. In more recent years, an International Work Group sponsored by the National Diabetes Data Group of the NIH proposed a now generally accepted classification, according to which the insulin-dependent ketosis-prone diabetes, formerly and inappropriately called the juvenile type, is considered a subclass of diabetes, type 1. Because it can occur at any age, it was recommended that the diagnosis based on age be eliminated. The non-insulin-dependent, non-keto sis-prone type of diabetes, which is not secondary to other diseases or conditions, and which was formerly called matu­ rity-onset diabetes, was considered a second subclass, type II, because although this form usually develops after age 40, it also occurs in young persons, who do not require insulin or are not ketotic. Although this classification is not entirely agreed upon by all diabetologists, for practical purposes it has been generally accepted and has been utilized by the contributors to this volume.

Keywords

Diabetes Diabetes mellitus Diabetic Pancreas Insulin Insulin Secretion Insulitis Islets Of Langerhans Pancreas Pancreatic Islets Viral Diabetes animals cancer cell microscopy pathology

Editors and affiliations

  • Bruno W. Volk
    • 1
  • Edward R. Arquilla
    • 1
  1. 1.University of California Irvine Medical CenterOrangeUSA

Bibliographic information