Acute Renal Failure in the Critically Ill

  • Rinaldo Bellomo
  • Claudio Ronco
Part of the Update in Intensive Care and Emergency Medicine book series (UICM, volume 20)

Table of contents

  1. Front Matter
    Pages I-XIII
  2. L. C. Racusen, K. Trpkov, K. Solez
    Pages 1-27
  3. J. P. Johnson, M. D. Rokaw
    Pages 28-57
  4. Z. Symon, M. Brezis
    Pages 58-63
  5. C. Tetta, G. Montrucchio, C. Ronco, G. Camussi
    Pages 64-89
  6. J. C. Lieske, F. G. Toback
    Pages 90-121
  7. A. D. Bersten, A. W. Holt
    Pages 122-146
  8. R. L. Lins, S. L. Chew, R. Daelemans
    Pages 147-159
  9. A. G. Fabris, G. Fabris
    Pages 165-177
  10. G. J. Kaloyanides
    Pages 178-214
  11. G. M. Chertow, W. F. Owen Jr., J. M. Lazarus
    Pages 265-297
  12. H. Kierdorf, H. G. Sieberth
    Pages 298-310
  13. R. Bellomo
    Pages 324-345
  14. C. Ronco, R. Bellomo
    Pages 364-384

About this book

Introduction

Over the last 10 years the syndrome of severe acute renal failure has progressively changed in its epidemiology. It is now most frequently seen in critically ill patients, typically in the context of sepsis and multiorgan failure. This epidemiologic change has meant that intensive care physicians and nephrologists must now work in close cooperation at all times and must take many com­ plex issues of prevention, pathogenesis, and management into account that they did not previously have to tackle. Simultaneously, the last 10 years have seen the development of major technical and conceptual changes in the field of renal replacement therapy. There are now previously unavailable therapeutic options that provide physicians with a flexible and rapidly evolving armamentarium. The nutrition of these patients, previously limited by the par­ tial efficacy of renal replacement therapies, has also become more aggressive and more in tune with the needs of critically ill patients. Increased understanding of the pathogenesis of the multi­ organ failure syndrome has focused on the role of many soluble "mediators of injury" (cytokines, leukotrienes, prostanoids etc.). These molecules are likely to participate in the pathogenesis of acute renal failure. Their generation and disposal is also affected by different techniques of artificial renal support.

Keywords

Assessment Drug-indicated renal failure Hemofiltration Heparorenal Syndrome Physiopathology Renal Failure care epidemiology immunology intensive care intensive care unit pathophysiology prevention shock

Editors and affiliations

  • Rinaldo Bellomo
    • 1
  • Claudio Ronco
    • 2
  1. 1.Intensive Care UnitAustin HospitalVictoriaAustralia
  2. 2.Dept. of Nephrology and Clinical DialysisSt. Bartolo HospitalVicenzaItaly

Bibliographic information

  • DOI https://doi.org/10.1007/978-3-642-79244-1
  • Copyright Information Springer-Verlag Berlin Heidelberg 1995
  • Publisher Name Springer, Berlin, Heidelberg
  • eBook Packages Springer Book Archive
  • Print ISBN 978-3-642-79246-5
  • Online ISBN 978-3-642-79244-1
  • Series Print ISSN 0933-6788