Abstract
The neocerebellar cortex lesion in newborn mice caused permanent asymmetry of the position of the hind limbs which was normalized spontaneously in about 27.8% of control animals. The administration of ribaminol, TCAP, and magnesium pemoline during the first 2 weeks of postnatal life did not improve the rate of recovery from the neocerebellar damage.
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This paper is sponsored by James Dyal, who takes full editorial responsibility for it. This work was supported by a grant from the Atkinson Charitable Foundation in Toronto. The author is indebted to Ruth Atkinson-Hindmarsh, President of the Foundation for support. Tricyanoaminopropene (U-9189) was obtained from the Upjohn Company of Canada through the courtesy of E. L. Masson. Pemoline magnesium was obtained from Abbott Laboratories, Scientific Division, North Chicago, 111. We are indebted to P. W. Dodge and A. O. Geiszler for the sample. We are also grateful to M. G. Stout, of the Nucleic Acid Research Institute, Irvine, Calif., for the sample of ribaminol. The author wishes to express his thanks to M. Reinis for her careful and reliable technical help.
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Reinis, S. Lack of effect of ribaminol, TCAP, and magnesium pemoline on recovery from neocerebellar damage. Bull. Psychon. Soc. 2, 385–386 (1973). https://doi.org/10.3758/BF03334421
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DOI: https://doi.org/10.3758/BF03334421