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Correction to: ZNF326 promotes malignant phenotype of glioma by up-regulating HDAC7 expression and activating Wnt pathway

  • Xinmiao Yu
  • Minghao WangEmail author
  • Jingjing Wu
  • Qiang Han
  • Xiupeng Zhang
Open Access
Correction
  • 66 Downloads

Correction to: J Exp Clin Cancer Res (2019) 38:40

https://doi.org/10.1186/s13046-019-1031-4

In the original publication of this manuscript [1], the author mislabeled the CTL group and ZNF326 group in Fig.2-I,J (MTT result). The revised Fig. 2 is shown below.
Fig. 2

Impact of ZNF326 expression on the proliferation and invasiveness of glioma cells in vitro. ZNF326 overexpression significantly enhanced the colony formation (a, b), invasiveness (e, f, magnification-400×), and proliferation (i, j) of U87 and U251 glioma cell lines. Conversely, ZNF326 knockdown significantly inhibited colony formation (c, d), invasiveness (g, h, magnification-400×), and proliferation (k, l) of U87 and U251 glioma cell lines. CTL: control group. Each experiment was performed in triplicate. Columns: mean numbers. Bars: S.D. (*: P < 0.05; **: P < 0.01; ***: P < 0.001)

The authors sincerely apologize for the inconvenience caused to the readers.

Reference

  1. 1.
    Yu X, et al. ZNF326 promotes malignant phenotype of glioma by up-regulating HDAC7 expression and activating Wnt pathway. J Exp Clin Cancer Res. 2019;38:40.  https://doi.org/10.1186/s13046-019-1031-4.CrossRefPubMedPubMedCentralGoogle Scholar

Copyright information

© The Author(s). 2020

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

Authors and Affiliations

  • Xinmiao Yu
    • 1
  • Minghao Wang
    • 2
    Email author
  • Jingjing Wu
    • 3
  • Qiang Han
    • 4
  • Xiupeng Zhang
    • 4
  1. 1.Department of Surgical Oncology and Breast SurgeryFirst Affiliated Hospital of China Medical UniversityShenyangChina
  2. 2.Department of NeurosurgeryFirst Affiliated Hospital of China Medical UniversityShenyangChina
  3. 3.Department of PathologyFirst Affiliated Hospital of Fujian Medical UniversityFuzhouChina
  4. 4.Department of Pathology, College of Basic Medical Sciencesand First Affiliated Hospital of China Medical UniversityShenyangChina

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