Abstract
THE cause of functional arrest of first set homotransplanted kidneys has recently been the subject of a series of biochemical investigations1–4. These investigations were begun to test the postulate that a primary biochemical lesion was responsible for the destruction of homo-transplanted kidneys5,6.
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TYLER, H., WILLIAMS, M. & DEMPSTER, W. Metabolism of Canine Renal Transplants. Nature 201, 84–85 (1964). https://doi.org/10.1038/201084a0
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DOI: https://doi.org/10.1038/201084a0
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