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High Shear Stress Marks: The Vulnerable Target with von Willebrand Factor for Platelet Deployment

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References

  1. Maalej N, Holden JE, Folts JD. Effects of shear stress on acute platelet thrombus formation in canine stenosed carotid arteries: An in vivo quantitative study. J Tromb & Thrombol 1998;5:231–238.

    Google Scholar 

  2. Kawahito K, Mohara J, Misawa Y, Fuse K. Platelet damage caused by the centrifugal pump: In vitro evaluation by measuring the release of alpha-granule packing proteins. Artif Organs 1997;21(10):1105–1109.

    Google Scholar 

  3. Mohara J, Kawahito K, Misawa Y, Fuse K. Evaluation of platelet damage in two different centrifugal pumps based on measurements of alpha-granule packing proteins. Artif Organs 1998;22(5):371–374.

    Google Scholar 

  4. Dintenfass L. Red cell aggregation in cardiovascular diseases and crucial role of inversion phenomenon. Angiology 1985;36:315–326.

    Google Scholar 

  5. Pace-Asciak, CR. Hepoxilin. Gen Pharmacol 1993;24:805–810.

    Google Scholar 

  6. Strony J, Beaudoin A, Brands D, Adelman B. Analysis of shear stress and hemodynamic factors in a model of coronary artery stenosis and thrombosis. Am J Physiol 1993; 265:H1787–1796.

    Google Scholar 

  7. Margalit A, Livne AA, Funder J, Granot Y. Initiation of RVD response in human platelets: Mechanical-biochemical transduction involves pertussis toxin sensitive G-protein and phospholipase A2. Memb Biol 1993;136:304–311.

    Google Scholar 

  8. Margalit A, Granot Y. Endogenous hepoxilin A3 produced under short duration of high shear-stress, inhibits thrombininduced aggregation in human platelets. Biochimica Biophysica Acta 1994;1900:173–176.

    Google Scholar 

  9. Margalit A, Gilutz H, Granot Y. Low regulatory volume decrease rate in platelets from ischemic patients: A possible role for hepoxilin A3. Platelets 1995;6:371–376.

    Google Scholar 

  10. Brune B, Hanstein K. Rapid reversibility of nitric oxide induced platelet inhibition. Thromb Res 1998;15:90(2):83–91.

    Google Scholar 

  11. Kroll MH, Hellurns JD, McIntire LV, Schafer AI, Maoke JL. Platelets and shear stress. Blood 1996;88:1525–1541.

    Google Scholar 

  12. Isaka N, Tanigawa T, Nishikawa M, Nakano T. High shear stress induced platelet aggregation and effects of antiplatelet therapy. Nippon Rinsho 1998;56(10):2624–2629.

    Google Scholar 

  13. Folts J. An in vivo model of experimental arterial stenosis, intimal damage, and periodic thrombosis. Circulation 1991; 83(Suppl 6):IV3–14.

    Google Scholar 

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  1. Cardiology Department, Soroka Medical Center and Faculty of Health Sciences, Ben-Gurion University, Beer-Sheva, Israel

    Harel Gilutz

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  1. Harel Gilutz
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Gilutz, H. High Shear Stress Marks: The Vulnerable Target with von Willebrand Factor for Platelet Deployment. J Thromb Thrombolysis 9, 251–252 (2000). https://doi.org/10.1023/A:1018718728589

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