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CCN3-mutant mice are distinct from CCN3-null mice

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Journal of Cell Communication and Signaling Aims and scope

Abstract

A recent manuscript reported phenotypic alterations associated to the expression of a CCN3 protein deleted for the Von Willebrand type C repeat that is common to the various members of the CCN family of proteins. In this comment, the biological significance of these alterations is briefly discussed.

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Authors and Affiliations

  1. Department of Dermatology, University of Michigan Medical School, Medical Science I, 1150 W. Medical Center Dr., Ann Arbor, MI, 48109-0609, USA

    B. Perbal

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  1. B. Perbal
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Correspondence to B. Perbal.

Additional information

This commentary also appears on line under Newsletter section of the ICCNS Website at http://ccnsociety.com

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Perbal, B. CCN3-mutant mice are distinct from CCN3-null mice. J. Cell Commun. Signal. 1, 229–230 (2007). https://doi.org/10.1007/s12079-008-0020-8

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  • DOI: https://doi.org/10.1007/s12079-008-0020-8

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