Der Nephrologe

, Volume 11, Issue 2, pp 150–152 | Cite as

Tubuloglomeruläres Feedback

Wie funktioniert es?
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Tubuloglomerular feedback

Notes

Interessenkonflikt

H. Castrop gibt an, dass kein Interessenkonflikt besteht.

Literatur

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    Bell PD, Lapointe JY, Sabirov R et al (2003) Macula densa cell signaling involves ATP release through a maxi anion channel. Proc Natl Acad Sci U S A 100:4322–4327PubMedCentralCrossRefPubMedGoogle Scholar
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    Castrop H, Huang Y, Hashimoto S et al (2004) Impairment of tubuloglomerular feedback regulation of GFR in ecto-5′-nucleotidase/CD73-deficient mice. J Clin Invest 114:634–642PubMedCentralCrossRefPubMedGoogle Scholar
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    Peterson OW, Gabbai FB, Myers RR et al (1989) A single nephron model of acute tubular injury: role of tubuloglomerular feedback. Kidney Int 36:1037–1044CrossRefPubMedGoogle Scholar
  4. 4.
    Sun D, Samuelson LC, Yang T et al (2001) Mediation of tubuloglomerular feedback by adenosine: evidence from mice lacking adenosine 1 receptors. Proc Natl Acad Sci U S A 98:9983–9988PubMedCentralCrossRefPubMedGoogle Scholar
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    Thurau K, Schnermann J (1965) The sodium concentration in the macula Densa cells as a regulating factor for Glomerular filtration (Micropuncture experiments). Klin Wochenschr 43:410–413CrossRefPubMedGoogle Scholar

Copyright information

© Springer-Verlag Berlin Heidelberg 2015

Authors and Affiliations

  1. 1.Institut für PhysiologieUniversität RegensburgRegensburgDeutschland

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