N-terminal domain of the RNA polymerase of very virulent infectious bursal disease virus contributes to viral replication and virulence
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This work was supported by the National Natural Science Foundation of China (31500129, 31430087).
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- Boot, H.J., ter Huurne, A.A.H.M., Hoekman, A.J.W., Peeters, B.P.H., and Gielkens, A.L.J. (2000). Rescue of very virulent and mosaic infectious bursal disease virus from cloned cDNA: VP2 is not the sole determinant of the very virulent phenotype. J Virol 74, 6701–6711.CrossRefPubMedPubMedCentralGoogle Scholar
- Hon, C.C., Lam, T.Y., Drummond, A., Rambaut, A., Lee, Y.F., Yip, C.W., Zeng, F., Lam, P.Y., Ng, P.T.W., and Leung, F.C.C. (2006). Phylogenetic analysis reveals a correlation between the expansion of very virulent infectious bursal disease virus and reassortment of its genome segment B. J Virol 80, 8503–8509.CrossRefPubMedPubMedCentralGoogle Scholar
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