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A novel sodium–glucose co-transporter 2 gene (SGLT2) mutation contributes to the abnormal expression of SGLT2 in renal tissues in familial renal glucosuria

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Acknowledgments

This work was funded by the National Natural Science Foundation of China (#81160087) and the Natural Science Fund of the Inner Mongolia Autonomous Region (#2010BS1102).

Conflict of interest

The authors declare that they have no conflict of interest.

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Authors and Affiliations

  1. Renal Division, Inner Mongolia People’s Hospital, Hohhot, 010017, People’s Republic of China

    Lei Yu & Guo-Ping Liu

  2. Renal Division, Peking University First Hospital, Peking University Institute of Nephrology, Key Laboratory of Renal Disease, Ministry of Health of China, Beijing, 100034, People’s Republic of China

    Ping Hou, Ji-Cheng Lv & Hong Zhang

Authors
  1. Lei Yu
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  2. Ping Hou
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  3. Ji-Cheng Lv
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  4. Guo-Ping Liu
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  5. Hong Zhang
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Corresponding author

Correspondence to Lei Yu.

Additional information

L. Yu and P. Hou are co-first authors and have contributed equally to the work.

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Yu, L., Hou, P., Lv, JC. et al. A novel sodium–glucose co-transporter 2 gene (SGLT2) mutation contributes to the abnormal expression of SGLT2 in renal tissues in familial renal glucosuria. Int Urol Nephrol 46, 2237–2238 (2014). https://doi.org/10.1007/s11255-014-0755-5

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  • DOI: https://doi.org/10.1007/s11255-014-0755-5

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