Experimental anatomic modeling of venous dyshemocirculation in the scrotal organs

Abstract

Disorders in venous outflow from the testis and epididymis lead to the formation of pathological compensatory hemodynamics, development of high intratesticular pressure with impairment of the integrity of the intra-organ vessels, up to extravasation (testicular venous infarction), to local arterial hypertension, inter-arterial shunting of stained solutions through the intersystem fusion of the testicular arteries (shunting of arterial blood under vital conditions), and hence, trigger the mechanism of secondary arterial ischemization of the testis and epididymis. The severity of circulatory disorders depends on the volume of venous collectors excluded from circulation: from pronounced disorders in case of testicular venous outflow blockade to extremely severe ones in case of combined testicular-cremasteric venous block (hemodynamic collapse).