References
Yamamoto F, Cid E, Yamamoto M, Blancher A (2012) ABO research in the modern era of genomics. Transfus Med Rev 26:103–118
Ogasawara K, Yabe R, Uchikawa M, Nakata K, Watanabe J, Takahashi Y, Tokunaga K (2001) Recombination and gene conversion-like events may contribute to ABO gene diversity causing various phenotypes. Immunogenetics 53:190–199
Yip SP (2002) Sequence variation at the human ABO locus. Ann Hum Genet 66:1–27
Verdiani S, Bonsignore A, Casarino L, Ferrari GM, Zia SC, De Stefano F (2009) ABO chimerism determined by real-time polymerase chain reaction analysis after ABO-incompatible haematopoietic stem cell transplantation. Int J Legal Med 123:431–435
Seltsam A, Wagner FF, Grüger D, Gupta CD, Bade-Doeding C, Blasczyk R (2007) Weak blood group B phenotypes may be caused by variations in the CCAAT-binding factor/NF-Y enhancer region of the ABO gene. Transfusion 47:2330–2335
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Initial funding for this work was supported by a grant from China Medical University. We thank BioMed Proofreading, LLC for assistance in English-language editing of the manuscript.
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The authors declare that they have no conflicts of interest.
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Pang, H., Li, C., Li, P. et al. A B101-O16 hybrid sequence is responsible for a Bweak phenotype in the ABO blood group. Int J Legal Med 129, 469–470 (2015). https://doi.org/10.1007/s00414-014-1052-7
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DOI: https://doi.org/10.1007/s00414-014-1052-7