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Conclusions/interpretation In rodent beta cell lines, we demonstrate hKLF11 overexpression-mediated inhibition of human proinsulin gene expression and characterise a prominent role for the CACCC box in maintaining basal proinsulin promoter activity.
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The online version of the original article can be found at http://dx.doi.org/10.1007/s00125-007-0667-3.
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Niu, X., Perakakis, N., Laubner, K. et al. Human Krüppel-like factor 11 inhibits human proinsulin promoter activity in pancreatic beta cells. Diabetologia 50, 2031 (2007). https://doi.org/10.1007/s00125-007-0770-5
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DOI: https://doi.org/10.1007/s00125-007-0770-5