Abstract
Resistance of plant pathogenic fungi towards QoI fungicides is mainly caused by two mutations in the cytochrome b gene, the F129L and G143A. In Pyrenophora teres, the causal agent of net blotch in barley, only the F129L mutation could be detected up to now. To investigate the practical impact of this mutation on the field efficacy of the QoI fungicide pyraclostrobin, field trials were performed on sites with different levels of F129L in populations of P. teres. Pyraclostrobin provided very good control of net blotch disease in fields with different frequency of the F129L in populations of P. teres in 2005 and 2006. The results suggest that the field performance of pyraclostrobin is not affected by the mutation F129L.
Zusammenfassung
Bei pflanzenpathogenen Pilzen ist die Resistenz gegenüber QoI-Fungiziden vor allem durch zwei Mutationen (F129L und G143A) im Cytochrom-b-Gen verursacht. In Isolaten von Pyrenophora teres, dem Erreger der Netzfleckenkrankheit der Gerste, konnte bisher nur die Mutation F129L nachgewiesen werden. Zur Untersuchung der Auswirkung dieser Mutation auf die Wirkung des QoI-Fungizids Pyraclostrobin unter Feldbedingungen wurden Feldversuche in den Jahren 2005 und 2006 auf Flächen mit unterschiedlich starker Ausbreitung der Mutation durchgeführt. Die Wirkung von Pyraclostrobin war auf allen Feldern sehr gut, unabhängig von der Häufigkeit der F129L-Mutation in der Population. Die Ergebnisse deuten darauf hin, dass die Feldwirkung von Pyraclostrobin gegen über P. teres durch die F129L-Mutation nicht beeinflusst wird.
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Semar, M., Strobel, D., Koch, A. et al. Field efficacy of pyraclostrobin against populations of Pyrenophora teres containing the F129L mutation in the cytochrome b gene. J Plant Dis Prot 114, 117–119 (2007). https://doi.org/10.1007/BF03356718
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DOI: https://doi.org/10.1007/BF03356718