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Wiggers, C. J. Cardiac Adaptions in Acute Progressive Anpxia. Ann. Int. Med.14 (1): 1237–1247 (1941).
Little, W. J. On the Nature and Treatment of the Deformities of the Human Frame. London: Longman, Brown, Green, and Longrinan (1853).
Osler, W. The Cerebral Palsies of Childhood, a Clinical Study from the Infirmary for Nervous Diseases. Philadelphia: P. Blaskiston (1889).
Freud, S. Die infantile Cerebrallähmung. Wien: Alfred Holder (1897).
Beecher, H. K., &Todd, D. P. A Study of Deaths Associated with Anesthesia and Surgery Based on a Study of 599,548 Anesthesias in Ten Institutions, 1948-1952, Inclusive. Ann. Surg.140 (1): 2–35 (1954).
Ford, F. R. Cerebral Birth Injuries and their Results. Medicine5 (5): 121–194 (1926).
Penfield, W., &Erickson, T. Epilepsy and Cerebral Localization: A Study of the Mechanism, Treatment, and Prevention of Epileptic Seizures. Springfield, Ill.: Charles C. Thomas (1941).
Nielsen, J. M. Etiology of Idiopathic Epilepsy. Bull. Los Angeles Neurol. Soc.11 (3): 97–101 (1946).
Nielsen, J. M., &Butler, F. O. Birth Primacy and Idiopathic Epilepsy. Bull. Los Angeles Neurol. Soc.13 (3): 176–178 (1948).
Nielsen, J. M., &Courville, Cyril B. Role of Birth Injury and Asphyxia in Idiopathic Epilepsy. Neurology1 (1): 48–52 (1951).
Courville, Cyril B. Narcosis and the Fetal brain. Bull. Los Angeles Neurol. Soc.20 (3): 97–111 (1955).
Benda, C. E. Developmental Disorders of Mentation and Cerebral Palsies. New York: Grune & Stratton (1952).
Fuldner, R. V. Labor Complications and Cerebral Palsy. Am. J. Obstet. & Gynec.74 (1): 159–166 (1957).
Windle, W. F., Becker, R. F., &Weil, A. Alterations in Brain Structure after Asphyxiation at Birth: An Experimental Study in the Guinea Pig. J. Neuropath. & Exper. Neurol.3 (4): 224–238 (1944).
Courville, Cyril B. Antenatal and Paranatal Circulatory Disorders as a Cause of Cerebral Damage in Early Life. J. Neuropath. & Exper. Neurol.18 (1): 115–139 (1959).
Courville, Cyril B., &Myers, R. O. Effects of Extraneous Poisons on the Nervous System. III. The Asphyxiant Gases. Bull. Los Angeles Neurol. Soc.19 (4): 197–225 (1954).
Courville, Cyril B. Case Studies in Cerebral Anoxia. IX. The Cerebral Lesion-Complexes Incident to Carbon Monoxide Asphyxia. Bull. Los Angeles Neurol. Soc.20 (3): 139–144 (1955).
Courville, Cyril B. Syndrome of Decorticate Rigidity, Convulsions, and Amentia Occurring in Early Infancy: Review of the Literature and Report of Four Verified Cases with Subtotal Softening of the Forebrain. Bull. Los Angeles Neurol. Soc.25 (1): 1–17 (1960).
Courville, Cyril B. Structural Alterations in the Cerebellum in Cases of Cerebellar Palsy: Their Relation to Residual Symptomatology in the Ataxic-Atonic Group. Bull. Los Angeles Neurol. Soc.24 (3): 148–165 (1959).
Courville, Cyril B. Pathogenesis of Nodular Atrophy of the Cerebral Cortex: A Common Cortical Change Found in Cases of Cerebral Palsy. Arch. Pediat.77 (3): 101–129 (1960).
Courville, Cyril B. The Pathogenesis of Nodular Cortical Atrophy: Apparent Mechanism of Lesion Commonly Found in Cerebral Palsied Individuals. Bull. Los Angeles Neurol. Soc.22 (3): 120–130 (1957).
Bresler, J. Klinische und pathologisch-anatomische Beiträgef zur Microgyrie. Arch. Psychiat.31 (1): 566–573 (1898-1899).
Morrison, L. R. Anatomical Studies of Central Nervous System of Dogs without Forebrain or Cerebellum. Haarlem: Bonin (1928). Cited by Benda (1952).
Malamud, N. Status marmoratus: A Form of Cerebral Palsy Following either Birth Injury or Inflammation of the Central Nervous System. J. Pediat.37 (10): 610–619 (1950).
Courville, Cyril B. Residual Lesions after Thrombosis of tne Superior Longitudinal Sinus: Review of Literature and Report of Case. Bull. Los Angeles Neurol. Soc.23 (4): 160–170 (1958).
Courville, Cyril B. Central Hemorrhagic Encephalopathy of Early Infancy: Report of Three Verified Cases Suggesting the Genesis of Infantile Cystic Degeneration in a Paranatal Anoxic Disorder. Neurology10 (1): 70–80 (1960).
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Much of the investigative work on which the present survey is based has been aided by a grant (B-1248) from the National Institute of Neurological Diseases and Blindness, Bethesda, Maryland.
In order to avoid confusion, the terms hypoxia and anoxia are used in the sense suggested by Wiggers.1 Accordingly, hypoxia implies a lowered oxygen tension in the blood resulting in physiological changes which are reversible leading to complete normalcy; anoxia indicates a state of oxygen lack to a degree resulting in pathological alterations in the brain followed by either fatal issue or physical and/or mental deficits during the survival period.
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Courville, C.B. Paranatal anoxia and its residual encephalic lesions. Can. Anaes. Soc. J. 8, 3–13 (1961). https://doi.org/10.1007/BF03015386
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DOI: https://doi.org/10.1007/BF03015386