References
Beckner, S. K.; Maluish, A. E.; Longo, D. A. Lymphokine-activated killer cells: culture conditions for the generation of maximal in vitro cytotoxicity in cells from normal donors. Cancer Res. 47:5504–5508; 1987.
Bell, E.; Ivarsson, B.; Merrill, C. Production of a tissue-like structure by contraction of collagen lattices by human fibroblasts of different proliferative potential in vitro. Proc. Natl. Acad. Sci. USA 76:1274–1278; 1979.
Buttle, D. J.; Ehrlich, H. P. Comparative studies of collagen lattice contraction utilizing a normal and a transformed cell line. J. Cell. Physiol. 116:159–166; 1983.
Clark, R. A. F.; Folkvord, J. M.; Hart, C. E., et al. Platelet isoforms of platelet-derived growth factor stimulate fibroblast to contract collagen matrices. J. Clin. Invest. 84:1036–1040; 1989.
Concannon, M. J.; Barret, B. B.; Adelstein, E. H., et al. The inhibition of fibroblast proliferation by a novel monokine: an in vitro and in vivo study. J. Burn Care Rehabil. 14:141–147; 1993.
Ehrlich, H. P.; Wyler, D. J. Fibroblast contraction of collagen lattices in vitro: inhibition by chronic inflammatory cell mediators. J. Cell. Physiol. 116:345–351; 1983.
Franzén, L.; Ghassemifar, R. Connective tissue repair in zinc deficiency. An ultrastructural morphometric study in perforated mesentery in rats. Eur. J. Surg. 158:333–337; 1992.
Franzén, L.; Norrby, K. A tissue model for quantitative studies on time course of healing, rate of healing, and cell proliferation after wounding. Acta Pathol. Microbiol. Scand. Sect. A Pathol. 91:281–289; 1983.
Franzén, L. E.; Schultz, G. S. Transforming growth factor-β enhances connective tissue repair in perforated rat mesentery but not peritoneal macrophage chemotaxis. Wound Rep. Reg. 1:149–155; 1993.
Leibovich, S. J.; Ross, R. The role of the macrophage in wound repair. Am. J. Pathol. 78(1):71–91; 1975.
Malcherek, P.; Franzén, L. A new model for the study of angiogenesis in connective tissue repair. Microvasc. Res. 42:217–223; 1991.
Montesano, R.; Orci, L. Transforming growth factor-β stimulates collagen matrix contraction by fibroblasts: implications for wound healing. Proc. Natl. Acad. Sci. USA 85:4894–4897; 1988.
Muul, L. M.; Nason-Burchenal, K.; Hyatt, C., et al. Studies of serum-free culture medium in the generation of lymphokine activated killer cells. J. Immunol. Methods 105:183–192; 1987.
Rappolee, D. A.; Werb, Z. Macrophage-derived growth factors. Current topics in microbiology and immunology. 181:87–126; 1992.
Rittenberg, T.; Ehrlich, H. P. Free fatty acids and dialyzed serum alterations of fibroblast populated collagen lattice contraction. Tissue Cell 24(2):243–251; 1992.
Skalli, O.; Gabbiani, G. The biology of the myofibroblast relationship to wound contraction and fibrocontractive decease. In: Clark, R. A. F.; Henson, P. M., eds. The molecular and cellular biology of wound repair. London: Plenum Press; 1988:373–402.
Steinberg, B. M.; Smith, K.; Colozzo, M., et al. Establishment and transformation diminish the ability of fibroblasts to contract a native collagen gel. J. Cell Biol. 87:304–308; 1980.
Tingström, A.; Heldin, C. H.; Rubin, K. Regulation of fibroblast-mediated collagen contraction by platelet-derived growth factor, interleukin-1a and transforming growth factor-β1. J. Cell Sci. 102:315–322; 1992.
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Ghassemifar, M.R., Ghassemifar, N. & Franzén, L.E. Macrophage-conditioned medium without serum enhances collagen gel contraction. In Vitro Cell Dev Biol - Animal 31, 161–163 (1995). https://doi.org/10.1007/BF02639426
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DOI: https://doi.org/10.1007/BF02639426