References
Frohlich ED. Haemodynamics of hypertension. In: Genest J, Koiw E, Kuchel O, eds.Hypertension: Physiopathology and treatment. New York: McGraw-Hill Book Company, 1977. pp. 15–49.
Frohlich ED. Hemodynamic factors in the pathogenesis and maintenance of hypertension.Fed Proceed 1982;41:2400–2408.
Frohlich ED. The heart in hypertension. In: Genest J, Kuchel O, Hamet P, et al. eds.Hypertension: Physiopathology and treatment, 2nd ed. New York: McGraw-Hill Book Company, 1983. pp. 791–810.
Frohlich ED. Hemodynamics and other determinants in development of left ventricular hypertrophy: Conflicting factors in its regression.Fed Proceed 1983;42:2709–2715.
Leitschuh M, Chobanian A. Vascular changes in hypertension.Medical Clinics of North America (in press).
Folkow B. Physiological aspects of primary hypertension.Physiol Reviews 1982;62:347–504.
Frohlich ED. Newer antihypertensive drugs. In: Yu PN, Goodwin JF, eds.Progress in Cardiology, Vol. 12. Philadelphia: Lea & Febiger, 1984. pp. 265–281.
Frohlich ED. Antihypertensive therapy: Newer concepts and agents.Cardiology 1985;72:349–365.
Frohlich ED. Changes in hypertrophy by treatment in hypertension: Results of experimental research. In: Kaufman W. Bonner G, Lang I, et al. eds.Primary hypertension. Berlin/Heidelberg: Springer-Verlag, 1986. pp. 105–114.
Frohlich ED. Potential mechanisms explaining the risk of left ventricular hypertrophy.Am J Cardiol 1987;59:91A-97A.
Frohlich ED. The heart in hypertension. In: Rosenthal J, Chobanian AV, eds.Arterial hypertension, 2nd ed. Springer-Verlag (in press).
Frohlich ED, Tarazi RC: Is arterial pressure the sole factor responsible for hypertensive cardiac hypertrophy?Am J Cardiol 1979;44:959–963.
Tarazi RC, Frohlich ED. Is reversal of cardiac hypertrophy a desirable goal of antihypertensive therapy?Circulation 1987;75:I113-I117.
Weil JV, Chidsey CA. Plasma volume expansion resulting from interference with adrenergic function in normal man.Circulation 1968;37:54–61.
Dustan HP, Tarazi RC, Bravo EL. Dependence of arterial pressure on intravascular volume in treated hypertensive patients.N Engl J Med 1972;286:861–866.
Perry HM (Chairman). Recommendations for a national high blood pressure program data base for effective antihypertensive therapy. Report of Task Force I, DHEW Publication No. (NIH) 75-593, US Dept of Health, Education and Welfare, Bethesda, MD, 1973.
Frohlich ED. Diuretics: State of the art.J Hypertension (in press).
Tarazi RC, Frohlich ED, Dustan HP. Plasma volume changes with long-term beta-adrenergic blockade.Am Heart J 1971;82:770–776.
Dreslinski GR, Messerli FH, Dunn FG, et al. Hemodynamics, biochemical and reflexive changes produced by atenolol in hypertension.Circulation 1982;65:1365–1368.
Ventura HO, Frohlich ED, Messerli FH, et al. Cardiovascular effects and regional blood flow distribution associated with angiotensin converting enzyme inhibition (captopril) in essential hypertension.Am J Cardiol 1985;55:1023–1026.
Dunn FG, Oigman W. Ventura HO, et al. Enalapril improves systemic and renal hemodynamics and allows regression of left ventricular mass in essential hypertension.Am J Cardiol 1984;53:105–108.
Amodeo C, Kobrin I, Ventura HO, et al. Immediate and short-term hemodynamic effects of diltiazem in patients with hypertension.Circulation 1986;73:108–113.
Frohlich ED, Messerli FH, Dreslinski GR, et al. Long-term renal hemodynamic effects of nadolol in patients with essential hypertension.Am Heart J 1984;108:1141–1143.
Fleckenstein A. History of calcium antagonists.Circ Res 1983;51:I3-I16.
Vaghy PL, Williams JS, Schwartz A. Receptor pharmacology of calcium entry blocking agents.Am J Cardiol 1987;59:9A-17A.
Bevan JA, Bevan RD, Hwa JJ, et al. Calcium regulation invascular smooth muscle: Is there a pattern to its variability within the arterial tree?J Cardiovasc Pharmacol 1986;8(Suppl 8):571–575.
Olivari MT, Bartorelli C, Polese A, et al. Treatment of hypertension with nifedipine: A calcium antagonist agent.Circulation 1979;59:1056–1062.
Lund-Johansen P. Hemodynamic long-term effects of verapamil in essential hypertension at rest and during exercise.Acta Med Scand 1984;Suppl 681:109–115.
Natsume T, Kardon MB, Pegram BL, et al. Ventricular performance in SHR with regressed cardiac hypertrophy. (Submitted)
Pegram BL, Frohlich ED. Immediate systemic and regional hemodynamic effects of MK-421 converting enzyme inhibitor in conscious Wistar-Kyoto and spontaneously hypertensive rats. In: Rascher W, Clough D, Ganten D, eds.Hypertensive mechanisms of the spontaneously hypertensive rat as a model to study human hypertension. New York and Stuttgart: F K Schattauer, 1982. pp. 677–680.
Natsume T, Gallo A, Pegram BL, et al. Hemodynamic effects of prolonged treatment with diltiazem in conscious normotensive and spontaneously hypertensive rats.Clin Exper Hyper 1985;A7:1471–1479.
Kobrin I, Sesoko S, Pegram BL, et al. Reduced cardiac mass by nitrendipine is dissociated from systemic or regional haemodynamic changes in rats.Cardiovasc Res 1984;3:158–162.
Garavaglia GE, Nunez BD, Messerli FH, et al. Systemic and regional hemodynamic effects of nitrendipine in patients with essential hypertension. (Submitted)
Loutzenheiser R, Epstein M. Effects of calcium antagonists on renal hemodynamics. An editorial review.Am J Physiol 1985;249:F619-F629.
Isshiki T, Amodeo C, Messerli FH, Pegram BL, Frohlich ED. Diltiazem maintains renal vasodilation without hyperfiltration in hypertension: Studies in essential hypertensive man and the spontaneously hypertensive rat.Cardiovascular Drugs and Therapy (this issue).
Ventura HO, Messerli FH, Oigman W, et al. Immediate hemodynamic effects of a new calcium-channel blocking agent (nitrendipine) in essential hypertension.Am J Cardiol 1983;51:783–786.
Pedrinelli R, Fouad FM, Tarazi RC, et al. Nitrendipine, a calcium-entry blocker. Renal and humoral effects in human arterial hypertension.Arch Intern Med 1986;146:62–65.
Bauer JH. Renal protective effects of calcium antagonists.Cardiovascular Drugs and Therapy (this issue).
Schmieder RE, Messerli FH, Garavaglia GE, et al. Cardiovascular effects of verapamil in essential hypertension.Circulation (in press).
Gomez DM. Evaluation of renal resistances with special reference to changes in essential hypertension.J Clin Invest 1951;30:1143–1155.
Kobrin I, Pegram BL, Frohlich ED. Acute pressure increase and intrarenal hemodynamics in conscious WKY and SHR rats.Am J Physiol 1985;18:H1114-H1118.
Myers BD, Deen WM, Brenner BM. Effects of norepinephrine and angiotensin II on the determinants of glomerular ultrafiltration and proximal tubule fluid reabsorption in the rat.Circ Res 1975;37:101–110.
Hostetter TH, Olson JL, Rennke HG, et al. Hyperfiltration in remnant nephrons: A potentially adverse response to renal ablation.Am J Physiol 1981;241:F85-F93.
Hostetter TH, Rennke HG, Brenner BM. The case for intrarenal hypertension in the initiation and progression of diabetic and other glomerulopathies.Am J Med 1982;72 375–380.
Zusman RM. Renin and non-renin-mediated antihypertensive action of converting enzyme inhibitors.Kidney Int 1984;25:969–983.
Navar LG, Rosivall L. Contribution of the renin-angiotensin system to the control of intrarenal hemodynamics.Kidney Int 1984;25:857–868.
Anderson S, Meyer TW, Rennke HG, et al. Control of glomerular hypertension limits glomerular injury in rats with reduced renal mass.J Clin Invest 1985;76:612–619.
Anderson S, Rennke HG, Brenner BM. Therapeutic advantage of converting enzyme inhibitors in arresting progressive renal diseases associated with systemic hypertension in the rat.J Clin Invest 1986;77:1993–2000.
Reams GP, Bauer JH. Effect of enalapril in subjects with hypertension associated with moderate to severe renal dysfunction.Arch Intern Med 1986;146:2145–2148.
Tagama Y, Kitamoto Y, Futaki G, et al. Effect of captopril on heavy proteinuria in azotemic diabetic patients.N Engl J Med 1985;313:1617–1620.
Author information
Authors and Affiliations
Rights and permissions
About this article
Cite this article
Frohlich, E.D. Correction of physiological alterations of hypertension. Cardiovasc Drug Ther 1, 345–348 (1987). https://doi.org/10.1007/BF02209074
Issue Date:
DOI: https://doi.org/10.1007/BF02209074