Advertisement

Function of the motor apparatus of the stomach in experimental lead intoxication

  • A. A. Mambeeva
Pathological Physiology and General Pathology
  • 19 Downloads

Summary

It was established in chronic experiments on dogs that the picture of experimental lead poisoning was to a considerable extent similar to that seen during clinical manifestations of saturnism in human beings. This similarity was manifested by the intermittent character of its course, i.e., repeated changes of aggravation (correspondingly “exacerbation”) and amelioration (correspondingly “lead carrier state”) periods. Registration of the motor function of the stomach in these animals indicated that its disturbances in chronically intoxicated animals also had a course with alternating periods of aggravation and normalization. Obvious lead intoxication was also accompanied by a marked depression of the motor function of the stomach.

Keywords

Internal Medicine Clinical Manifestation Motor Function Considerable Extent Carrier State 
These keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.

Preview

Unable to display preview. Download preview PDF.

Unable to display preview. Download preview PDF.

Literature Cited

  1. 1.
    S. Ya. Barenblat and N. S. Pravdin, In the book: Prevention of Lead and Industrial Poisoning [in Russian], Moscow (1935), p. 152.Google Scholar
  2. 2.
    P. G. Bogach, Mechanisms of Nervous Regulation of the Motor Function of the Small Intestine [in Russian], Kiev (1961).Google Scholar
  3. 3.
    V. N. Boldyrev, Periodic Activity of the Digestive Apparatus in the Empty Stomach [in Russian], Thesis SPB (1904).Google Scholar
  4. 4.
    D. M. Brener, Condition of the Gastrointestinal Tract in Chronic Lead Poisoning [in Russian], Thesis, Alma-Ata (1957).Google Scholar
  5. 5.
    O. S. Glozman, A. I. Zikeeva, and M. S. Saulebekova, Nauchn. Izv. Kazakhsk. Med. Inst. No. 15, 70 (1958).Google Scholar
  6. 6.
    F. V. Grinberg, Gig. Truda, No. 11, 40 (1927).Google Scholar
  7. 7.
    F. V. Grinberg,Ibid., No. 2 14 (1928).Google Scholar
  8. 8.
    K. F. Elenevskii, In the book: Studies on Lead Poisoning [in Russian], Khar'kov (1926), p. 123.Google Scholar
  9. 9.
    T. N. Kaliteevskaya, Sov. Med., No. 4, 96 (1957).Google Scholar
  10. 10.
    A. A. Mambeeva, Izv. AN Kazakhsk. SSR, Seriya Med. i Fiziol., No. 1, 87 (1959).Google Scholar
  11. 11.
    A. A. Mambeeva,Ibid., No. 2, 66 (1960).Google Scholar
  12. 12.
    A. A. Mambeeva, Byull. Éksp. Biol., No. 4, 41 (1963).Google Scholar
  13. 13.
    A. L. Morozov, Findings on the Functional State of the Digestive Glands in Some Disorders (Anemia, Ulcers, Poisoning, Silicosis) [in Russian], Thesis, Moscow (1953).Google Scholar
  14. 14.
    V. F. Mostun, Byull. Éksp. Biol.,29, No. 6, 410 (1950).Google Scholar
  15. 15.
    I. P. Razenkov, Selected Works [in Russian], Moscow (1959), p. 202.Google Scholar
  16. 16.
    I. P. Razenkov, Selected Works [in Russian], Moscow (1959), p. 212.Google Scholar
  17. 17.
    I. P. Razenkov, Selected Works [in Russian], Moscow (1959), p. 218.Google Scholar
  18. 18.
    N. A. Senkevich, State of the Gastric Secretion in Some Occupational Disorders (Silicosis, Lead and Mercury Poisoning) [in Russian], Thesis, Moscow (1953).Google Scholar
  19. 19.
    M. I. Stychinskaya, Trudy Inst. Kraevoi Patologii AN Kazakhsk. SSR. Alma-Ata,9, 141 (1961).Google Scholar
  20. 20.
    I. Aub, L. Fairhall, A. Minot et al., Lead Poisoning, Baltimore (1926).Google Scholar
  21. 21.
    T. Wassermann, Arch. Exp. Path. Pharm., Bd. 79, S. 383 (1916).Google Scholar

Copyright information

© Consultants Bureau 1966

Authors and Affiliations

  • A. A. Mambeeva
    • 1
    • 2
  1. 1.Kazakh Institute of Marginal ParthologyAcademy of Medical Sciences of the USSRAlma-Ata
  2. 2.Laboratory of Digestive Pathology and Physiology Institute of Normal and Pathological PhysiologyAcademy of Medical Sciences of the USSRMoscow

Personalised recommendations