Changes in glycogenolysis in the zone of ischemia in experimental myocardial infarction
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During the first minutes after occlusion of the coronary artery in dogs, increased glycogenolysis was detected, for which activation of phosphorylase, phosphofructokinase, triose phosphate isomerase, and lactate dehydrogenase was responsible; aldolase and glyceraldehyde-3-phosphate dehydrogenase reactions became the stages limiting the rate of glycogenolysis. Activation of glycogenolysis was evidently due to the combined action of hypoxia and catecholamines.
Key Wordsexperimental myocardial infarction glycogenolysis
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