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Qualitative erythrocyte enzymatic disorder in Ehrlich tumor-bearing mice

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Summary

Erythrocyte glutathione reductase Michaelis-Menten constant was found to be significantly increased in Ehrlich tumour bearing mice, indicating a clear qualitative disturbance due to a neoplastic process. No changes were observed in glucose-6-phosphate dehydrogenase.

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References

  • Barretto OCO, Zyngier, S (1984) Acquired disturbance of erythrocyte glutathione reductase in experimental tumors. J Cancer Res Clin Onc 108:252–254

    Google Scholar 

  • Beutler E (1975) Red cell metabolism. A manual of biochemical methods. 2nd. ed., Grune & Stratton, New York, pp 66–71

    Google Scholar 

  • Boivin P, Galand C, Hakin J, Kahn A (1975) Acquired erythroenzymopathies in blood disorders: study of 200 cases. Br J Hematol 31:531–544

    Google Scholar 

  • Kahn A (1981) Abnormalities of eryhtrocyte enzymes in diserythropoiesis and malignancies. Clin Hematol 10:123–138

    Google Scholar 

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This work was performed in the Laboratório de Pesquisas Hematológicas (Prof. Dr. Michel Jamra) and in the Laboratório de Terapêutica Clinica, Faculdade de Medicina da Universidade de São Paulo

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Barretto, O.C.O., Zyngier, S.B. Qualitative erythrocyte enzymatic disorder in Ehrlich tumor-bearing mice. J Cancer Res Clin Oncol 110, 247–248 (1985). https://doi.org/10.1007/BF00399283

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  • DOI: https://doi.org/10.1007/BF00399283

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