Catecholamines, α-, and β-Adrenoceptors Are Not Responsible for Activation of Duodenum Motility Induced by Sympathetic Nerve Stimulation
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Stimulation of sympathetic nerve in anesthetized dogs not treated with adrenergic blockers more frequently exerted stimulating rather than inhibitory effect on duodenal motility. Blockade of α- and β-adrenoceptors with phentolamine and propranolol, respectively, did not prevent the excitatory action of the sympathetic nerve stimulation, but even potentiated this effect. The data showed that catecholamines as well as α- and β-adrenoceptors are not involved in the excitatory effect of sympathetic origin.
Key Words
duodenum neural control catecholamines α- and β-adrenoceptorsPreview
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