Knee Surgery, Sports Traumatology, Arthroscopy

, Volume 20, Issue 7, pp 1349–1356 | Cite as

Length-change patterns of the collateral ligaments after total knee arthroplasty

  • K. Milton Ghosh
  • Azhar M. Merican
  • Farhad Iranpour
  • David J. Deehan
  • Andrew A. Amis
Knee

Abstract

Purpose

Total knee arthroplasty (TKA) is a procedure with function dependent upon correct tensioning of the soft-tissue constraints. The purpose of this study was to examine the length-change behaviour of the collateral ligaments during knee flexion–extension before and after TKA. The influence of differing degrees of internal-external rotation of the femoral component on slackening/tightening of the collateral ligaments during knee flexion was to be studied.

Methods

The length-change patterns of the collateral ligaments were measured in eight intact knees in vitro: sutures were passed along the ligaments and attached to displacement transducers. Measurements were repeated after TKA with the femoral component in neutral rotation, then with 5° internal and 5° external rotation.

Results

Both the MCL and LCL slackened during knee flexion from 0° to 110° flexion, at all stages of the experiment. In the native knee, the MCL slackened 2 mm, whilst the LCL slackened 7 mm. The MCL slackened a further 3 mm and the LCL a further 4 mm during flexion post-TKA. A 5° external rotation of the femoral component slackened the MCL 2 mm more and tightened the LCL by 2 mm. The opposite effects resulted from 5° internal rotation.

Conclusions

The collateral ligaments slackened more than normal following TKA, and these length changes were increased by femoral component rotation. External rotation of the femoral component to address patellar tracking may slacken the MCL and thus lead to valgus instability in the flexed knee.

Keywords

Total knee arthroplasty Collateral ligaments Length changes Femoral component rotation Biomechanics in vitro 

Notes

Acknowledgments

The running costs of this study were supported by Smith and Nephew (Reconstruction) UK. Azhar M. Merican was supported by Arthritis Research (UK) and the University of Malaya Medical Centre, Kuala Lumpur.

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Copyright information

© Springer-Verlag 2011

Authors and Affiliations

  • K. Milton Ghosh
    • 1
    • 2
  • Azhar M. Merican
    • 3
    • 4
  • Farhad Iranpour
    • 4
  • David J. Deehan
    • 1
  • Andrew A. Amis
    • 2
    • 4
  1. 1.Orthopaedic Surgery DepartmentNewcastle University HospitalNewcastle upon TyneUK
  2. 2.Mechanical Engineering DepartmentImperial College LondonLondonUK
  3. 3.Department of Orthopaedic SurgeryUniversity of Malaya Medical CentreKuala LumpurMalaysia
  4. 4.Orthopaedic Surgery Group, Department of Surgery and Cancer, Imperial College London School of MedicineCharing Cross HospitalLondonUK

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