Inflammation Research

, Volume 64, Issue 11, pp 841–844 | Cite as

Nurr1 reduction influences the onset of chronic EAE in mice

  • Francesca Montarolo
  • Simona Perga
  • Serena Martire
  • Antonio Bertolotto
Short Communication

Abstract

Objective

Nurr1 plays anti-inflammatory functions in astrocytes/microglia. Gene expression analysis reveals Nurr1 down-regulation in PBMCs of MS patients that negatively correlates with disease aggressiveness. This study assesses the consequences of Nurr1 reduction in a MS model represented by EAE.

Methods

EAE was induced in heterozygous Nurr1 knockout mice. Clinical course was evaluated during pre-symptomatic, acute, and chronic phases. Neurohistopathological state was analyzed in spinal cord.

Results and conclusions

Nurr1 defect induces early EAE onset and increases inflammatory infiltrates in spinal cord suggesting a Nurr1 role in the early phase of EAE.

Keywords

Nurr1 Multiple sclerosis Experimental autoimmune encephalomyelitis Inflammation 

Notes

Acknowledgments

The Nurr1-KO mice were originally from the laboratory of Prof. O. M. Conneely (Baylor College Medicine, Houston, USA). The work was supported by the Fondazione Italiana Sclerosi Multipla (FISM 2010/R/7).

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Copyright information

© European Union 2015

Authors and Affiliations

  • Francesca Montarolo
    • 1
  • Simona Perga
    • 1
  • Serena Martire
    • 1
  • Antonio Bertolotto
    • 1
  1. 1.AOU San Luigi Gonzaga, Neurobiology UnitNeurologia 2, CReSM (Regional Referring Center of Multiple Sclerosis) and Neuroscience Institute Cavalieri Ottolenghi (NICO)OrbassanoItaly

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