Permanent inferior binasal quadrantanopsia in pseudotumor cerebri

  • Nichelli P. 
  • Penne A. 
Case Reports

Abstract

A sixty-year old patient had inferior nasal visual field defect in both eyes associated with papilledema. After pseudotumor cerebri was diagnosed and the appropriate treatment begun, the papilledema disappeared, but the visual field defect persisted.

This case suggests a revision of the classical concepts concerning the pathogenesis of binasal defects from intracranial causes and emphasizes the risk of loss of vision inherent in long-standing papilledema.

Key-words

Inferior nasal quadrantopsia pseudotumor cerebri chronic papilledema 

Sommario

Viene descritto il caso di un paziente di 60 anni con una quadrantanopsia binasale inferiore e papil-ledema dovuti a pseudotumor cerebri. Dopo l'inizio della opportuna terapia si è osservata una regressione del papilledema ma non del deficit campimetrico.

Questo caso suggerisce una revisione della concezione classica sulla patogenesi dei difetti binasali da patologia intracranica e sottolinea i rischi di danno visivo inerenti un papilledema di lunga durata.

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References

  1. [1]
    Corbett J.J., Savino P.J., Thompson H.S. et al.:Visual loss in pseudotumor cerebri. Follow-up of 57 patients from five to 41 years and a profile of 14 patients with permanent severe visual loss. Arch. Neurol. 39: 461–474, 1982.PubMedGoogle Scholar
  2. [2]
    Cushing H., Walker C.B.:Distorsion of the visual fields in cases of brain tumor. Binasal hemianopsia. Arch. Ophthalmol. 41: 559–598, 1912.Google Scholar
  3. [3]
    Dersh J., Sclezinger N.S.:Inferior nasal quadrantanopsia in pseudotumor cerebri. Arch. Neurol. 1: 695–699. 1959.Google Scholar
  4. [4]
    Lutz A.:Ueber binasale hemianopsie. V. Graefe's Arch. Opht. 119: 423–487; 1928.CrossRefGoogle Scholar
  5. [5]
    Minckler D.S., Bunt A.H., Klock I.B.:Radioautographic and cytochemical ultrastructural studies of axoplasmic transport in the monkey optic nerve head. Invest. Ophthalmol. Vis. Sci. 17:33–50, 1978.PubMedGoogle Scholar
  6. [6]
    Morse P.N., Leveille A.S., Antel J.P. et al:Bilateral juxtapapillary subretinal neovascularization associated with pseudotumor cerebri. Am. J. Ophthalmol. 91: 312–317, 1981.PubMedGoogle Scholar
  7. [7]
    O'connell J.E.A., Du Boulay P.G.H.:Binasal hemianopia. J. Neurol. Neurosurg. Psychiat. 36: 697–709, 1973.PubMedGoogle Scholar
  8. [8]
    Pilley S.F.J. Thompson H.S.:Binasal field loss and “pre-fixation blindness”. In: Neuro-ophthalmology. Vol. 8, pp. 277–284 (eds. Glaser J.S., Smith J.L.). St. Louis: C.V. Mosby Co., 1975.Google Scholar
  9. [9]
    Quigley H.A., Addick E.M.:Regional differences in the structure of the lamina cribrosa and their relation to glaucomatous optic nerve damage. Arch. Ophthalmol. 99: 137–143, 1980.Google Scholar
  10. [10]
    Salinas-Garcia R.F., Smith J.L.:Binasal hemianopia. Surg. Neurol. 10:187–194, 1978PubMedGoogle Scholar

Copyright information

© Masson Italia Editori S.p.A. 1983

Authors and Affiliations

  • Nichelli P. 
    • 1
  • Penne A. 
    • 2
  1. 1.Clinica Neurologica dell'Università di ModenaItalia
  2. 2.Clinica Oculistica dell'Università di ModenaItalia

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